Monday, April 29, 2013

Human Genetic Differences





 This article by Lloyd Pye tackles the one remaining conjecture regarding the emergence of modern humanity.

That conjecture has a cultural tradition behind it and additional geological support that has been largely ignored.

What it boils down to is that genetic intervention has occurred at least twice in human history. The first time occurred at least 200,000 years ago and possibly sooner. This led to the emergence of some form of modern man by 40,000 years ago.

The second event was the direct seeding of Earth with genetically prepared colonies of agricultural man along with the critical toolkit. This was done about 9000 BP with several large colonies afte4r the effects of the Pleistocene Nonconformity had settled down and the Holocene became well established.

This obviously means real engineering of the human genome way beyond any natural explanation. That is pretty evident anyway by simple inspection but also indicative of our next step. Just how does our genome differ? As it turns out, the differences are both radical and actually complete improbabilities using any natural protocol.

It goes without saying that every geneticist has the skills to disprove the general conjecture by reviewing the data and the claims. Instead their silence remains.

What I would like to see is this work replicated on the San, the Pygmies and the Bushmen in particular. They are the populations that may still have missing changes intact. Other populations should also be tested and cataloged against this understanding.

All other populations have long since been hybridized away.

The take home from this article is that our genome bears ample evidence of genetic manipulation whose natural provenance is a total impossibility.


What About Genetic Differences?



ye.com/interventionebook.html


Nothing supports the Intervention Theory and its Intragalactic Terraformers quite as much as the fascinating genetic differences between our human DNA, and chimp and gorilla DNA; and, since recent recovery, Neanderthal DNA; and, at some point in the future, hominoid DNA.

As we explained a few pages ago, the second chromosome in humans is a fusion of the 2nd and 3rd chromosomes in higher primates (HP).

The mainstream claims it was caused by a rare mutation called a Robertsonian translocation, which can combine chromosomes end-to-end, telomeres-to-telomeres, to somehow make them function well in a radically new configuration.

To combine two chromosomes so they can keep working is such an incredibly complex series of events, if it were not for mainstreamers having a desperateneed for that event to be considered plausible, they would laugh it out of existence.

Let’s try to follow their logic. Since all HP have 48 chromosomes (24 from each parent) it seems safe to assume any “common ancestor” (CA) of chimps and humans had 48 chromosomes.

Let’s assume two CAs have sex, and somehow in that process, the female’s egg has undergone a Robertsonian translocation mutation and its 2nd and 3rd chromosomes have fused into one.

When that mutated egg meets any normal, 24 chromosome sperm, it will not form a fertilized zygote . . . or if it does, soon after it will expire. Why? Because to replicate into more cells, each chromosome must line up with its pair from the other parent before being duplicated and pulled apart by fibers to opposite ends of the cell.



Then, the one cell splits into two. This process is called mitosis, and every one of our trillions of cells are copied in this manner, one after the other after the other, from the original one cell.

Now, if we consider a human-chimp cross, the 2nd human chromosome must line up with two chimp chromosomes. However, as the contents of the cell first duplicate and then pull apart, the intricate “dance” between them will soon end.
Why? One copy of the human 2nd chromosome and one copy each of the chimp 2nd and 3rd must somehow safely wind up at one end of the cell, while the other three copies need to be pulled to the other end before the cell can divide.

In that process the fibers become confused, and the resulting cells try to keep on replicating, but that chaos continues until the blastula expires.

So, there is no way a one-chromosome-short zygote will somehow become a viable fetus.



In addition to the above, now let’s consider the problems found in telomeres and centromeres.

Telomeres are the “caps” found at the ends of chromosomes that gradually reduce after each cell division. Think of them as a long string of “beads” on a necklace, and after each division of each body’s trillions of cells, a bead is lost.

When all of the telomeres have dropped off, the chromosomes stop replicating and the organism they support will die from advanced “old age.” Nothing can stop the slow loss of those beads.

Centromeres are segments of DNA usually located near a chromosome’s middle, and they are critical to successful cell division, which is the continual process of life that has to happen correctly, each time, every time, or things can go very, very haywire within the organism.

Now, with that in mind, let’s try to imagine what would happen if a pair of chromosomes fuse in the way the two primate chromosomes fused to create the “missing” one in humans.



The fusion puts the two central telomeres (blue) into the middle where the centromeres should be, and the new chromosome has a pair of (red) centromeres when it should only have one, and that one should be where the telomeres are.

This is a serious problem because telomeres perform a “stopping” function that is entirely inappropriate in the middle of a chromosome that is supposed to be fully functional. Uh-oh!

Even worse, the centromeres are only useful in cell division, so when that occurs there will be not one, but two places where it is happening, which will soon lead to a badly tangled mess.

Clearly, mainstreamers need multiple miracles to make this scenario plausible . . . and guess what? The exact array of miracles required has been found within human chromosome #2!

Traces of two HP telomeres are found in human chromosome #2, between bases #114,455,823 and #114,455,838. Those 15 are deactivated in some way that doesn’t stop the chromosome’s normal functioning. They have been neutered!

With the fused chromosomes, only the middle two telomeres are “deactivated.” The one at the top end and the bottom one at the other end are not altered, so their crucial role in cell division (dropping “beads”) will continue unhindered. How amazing is that? How . . . coincidental?

As for leaving two centromeres where only one can function, guess what? One of those seems also to have been deactivated, so that normal cell division can proceed successfully! Wow!

The sequential precision of this incredible, one-in-trillions fusion forces us to describe it as yet another of the many miracles the mainstream always seems to be blessed with. Incredible!

The Big Kahuna of Human Genetics

While the fusion “miracles” torture credibility for anyone except mainstreamers, believe it or not we find several more in other chromosomes in the human genome! These are inversions.

An inversion can occur when a segment of any chromosome is sliced into, top and bottom, and then pulled out, inverted, and put back into its original place, but with a “flipped” orientation.



According to textbooks, inversions are caused by “ionizing radiation” that causes the genetic bonds of chromosome’s to “temporarily” break loose, during which inversion occurs, followed by a reinsertion. These are rare, but verifiable.
Also consider that any two chromosomes might accidentally become “entangled,” and the result is the brief tearing loose of one segment that then inverts and moves back into its place.



The beauty of this is that every inversion is unique, and if passed on creates a landmark DNA signpost which cannot be reversed back to normal in future generations that carry it. It also works to disprove Darwin, as we shall see.

In theory, while an inversion changes the order of the alleles that comprise chromosomes and genes, the overall makeup of both will remain unharmed as long as every gene temporarily segregated from the chromosome is retained in the process of inversion and reinsertion.

Now, brace yourself for this: The genome of every human carries nine of those “miraculous” inversion/insertions that are not found in any of the corresponding chimp chromosomes! They are located in these human chromosomes:

1, 4, 5, 9, 12, 15, 16, 17, and 18!

According to Darwinian evolutionary gospel, this means that at some point after the proto-humans and chimps split from their supposed common ancestor, the first of the 9 inversions occurred, eventually to be followed by 8 more.

For example’s sake, let’s assume that the first occurred in chromosome #1, at 5 mya. One of the new proto-humans carrying those fused chromosomes gives birth to a child with an inversion/insertion not carried by chimps in chromosome #1. At 5 mya. Simple enough.

Now that child must run the gamut of infant and child mortalities to reach maturity. It does, and then finds a mate. Unlike the chromosome fusion case, which won’t allow offspring with a partner having a loose chromosome, inversions can be passed on with a partner who lacks it.

In each pairing, the offspring will have a one-in-two chance of inheriting the new inversion. The same will hold true for their offspring if they carry it, so their odds of passing it to any one of their children would be 50% - 50%.

Now imagine some astronomical odds. What the above means is that somehow the individual with the insertion in chromosome #1 produced a line of offspring that passed it down to every descendant member of its species to become a part of nearly 7 billion humans alive today!

Since today we all have an identical insertion in our #1 chromosome, it means the insertion had to start at some point with a mutation in one of us who somehow bequeathed it to the rest of us.

This mutation, whether it did something good for the individual who had it, or bad, or nothing at all, would, according to Darwinists, create an aberration that mushroomed out into humanity like a nuclear bomb. Now hold that thought.

For as unlikely as all that is, guess what? The mushroom cloud inversion improbability had to occur in exactly that way for every one of the eight more times it occurred! That’s right, it is, in fact, massive improbability to a power of 9!

In a few million years, on 9 separate occasions, proto-humans were born with a new and quite distinct inversion mutation that would then be passed on to everyhuman alive on Earth today. So the odds of that occurring are enormously more than long, they are beyond imagining!

With all that said, here is the kicker, the thing that will lay you low if you’re not ready for it: Each one had to happen in a sequence! If they all occurred together, it wouldn’t be evolution.

Let’s get clear on what the mainstream insists had to happen. Inversion #1 occurred and the genetic lines of all the other proto-humans had to die out. Only its progeny would live to pass the inversion along to subsequent generations.

Next, let’s say that at 4.5 mya the inversion in chromosome #4 occurred, and, of course, that one has to occur in one of the progeny in whom the first

Now only its progeny, carrying the inversions in chromosome #1 and #4, can move forward into the future. All other proto-human lines at 4.5 mya have to die out in one way or another.

If the mainstream is right, and the 9 randomly generated inversions occurred in a Darwinian sequence of gradually accumulating mutations, it couldn’t happen any other way. However, is there another way? One that makes more sense?
Of course there is!

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